http://researchonline.federation.edu.au/vital/access/manager/Index ${session.getAttribute("locale")} 5 http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14820 Wed 25 Nov 2020 14:15:41 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14790 Wed 07 Apr 2021 14:02:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14777 Wed 07 Apr 2021 14:02:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14683 Wed 07 Apr 2021 14:02:46 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14609 Wed 07 Apr 2021 14:02:42 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14599 Wed 07 Apr 2021 14:02:41 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14607 Wed 07 Apr 2021 14:02:41 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14580 Wed 07 Apr 2021 14:02:40 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14583 Wed 07 Apr 2021 14:02:40 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14564 Wed 07 Apr 2021 14:02:39 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14469 Wed 07 Apr 2021 14:02:33 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14277 Wed 07 Apr 2021 14:02:22 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14162 Wed 07 Apr 2021 14:02:14 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14048 Wed 07 Apr 2021 14:02:08 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13980 Wed 07 Apr 2021 14:02:04 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13808 Wed 07 Apr 2021 14:01:55 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13780 0.1). The depressor response to pentolinium was greater in BPH/2J than BPN/3J, but in both cases the response in RD mice was similar to sham. Enalaprilat decreased MAP more in RD BPH/2J compared to sham (−12 vs −3 mmHg, P < 0.001) but had no effect in BPN/3J. RD reduced renal noradrenaline in both strains but more so in BPH/2J. RD reduced renin mRNA and protein, but not plasma renin in BPH/2J to levels comparable with BPN/3J mice. We conclude that renal nerves contribute to hypertension in BPH mice as RD induced a sustained fall in MAP, which was associated with a reduction of intrarenal renin expression. The lack of inhibition of the depressor effects of pentolinium and enalaprilat by RD suggests that vasoconstrictor effects of the SNS or RAS are not involved.]]> Wed 07 Apr 2021 14:01:53 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13695 Wed 07 Apr 2021 14:01:48 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13530 Wed 07 Apr 2021 14:01:39 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13501 Wed 07 Apr 2021 14:01:38 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13492 Wed 07 Apr 2021 14:01:37 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13349 Wed 07 Apr 2021 14:01:30 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13098 Wed 07 Apr 2021 14:01:17 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13085 Wed 07 Apr 2021 14:01:16 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13060 Wed 07 Apr 2021 14:01:15 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13001 Wed 07 Apr 2021 14:01:11 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12928 15,500 cases and > 500 deaths. Methods: Using the national cholera database, we analysed the spatio-temporal distribution and clustering of the Papua New Guinea cholera outbreak. The Kulldorff space-time permutation scan statistic, contained in the software package SatScan v9.2 was used to describe the first 8 weeks of the outbreak in Morobe Province before cholera cases spread throughout other regions of the country. Data were aggregated at the provincial level to describe the spread of the disease to other affected provinces. Results: Spatio-temporal and cluster analyses revealed that the outbreak was characterized by three distinct phases punctuated by explosive propagation of cases when the outbreak spread to a new region. The lack of road networks across most of Papua New Guinea is likely to have had a major influence on the slow spread of the disease during this outbreak. Conclusions: Identification of high risk areas and the likely mode of spread can guide government health authorities to formulate public health strategies to mitigate the spread of the disease through education campaigns, vaccination, increased surveillance in targeted areas and interventions to improve water, sanitation and hygiene.]]> Wed 07 Apr 2021 14:01:08 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12914 7 mg/dL in men and >6 mg/dL in women. The association between sUA concentration and SBP and DBP was examined using Pearson's correlation test, multivariate linear regression, and logistic regression analysis. The prevalence of hypertension and HUA increased with age (P < .001). Hypertension was more common in participants that had HUA than in those that did not (38.95% vs 30.16%, P = .02). Higher sUA was significantly associated with higher SBP and DBP in the 41- to 50-year-old participants (SBP, β = 0.35, P < .001; DBP, β = .29, P < .001; after adjustment for age, sex, total cholesterol, estimated glomerular filtration rate, and fasting plasma glucose). HUA was also a risk factor for hypertension in this age group (odds ratio 1.425, 95% confidence interval, 1.217-1.668, P < .001). There was no association between sUA concentration and SBP and DBP in the other age groups. In this population of healthy Chinese participants, sUA concentration was positively associated with hypertension only in the 41- to 50-year-old group. Lowering uric acid in this age group may help to reduce the incidence of hypertension.]]> Wed 07 Apr 2021 14:01:07 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12740 Wed 07 Apr 2021 14:00:57 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12267 Wed 07 Apr 2021 14:00:33 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11954 Wed 07 Apr 2021 13:57:14 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11922 Wed 07 Apr 2021 13:57:12 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11795 0.04 ng/mL), after performing high-intensity cycle ergometer sprints. As creatine kinase (CK) is expressed by multiple organs (e.g., skeletal muscle, brain, and myocardium), cTnI assays were performed to determine any changes in total serum CK levels not originating from skeletal muscle damage. Diagnosis: A posteriori the individual's daily energy expenditure indicated chronically low-energy availability. Psychometric testing suggested that the individual scored positive for disordered eating, highly for fatigue levels, and low in mental health components. Outcomes: The current case report provides novel evidence of elevated cTnI occurring as a result of performing short duration, high intensity, cycle ergometer exercise in an individual with self-reported chronically depleted energy balance. A schematic to identify potentially “at risk” individuals is presented. Lessons: Considering this as a case report, results cannot be generalized; however, the main findings suggest that individuals who habitually restrict their calorie intake below their bodies’ daily energy requirements, may have elevated biomarkers of exercise induced myocardial stress from performing high-intensity exercise.]]> Wed 07 Apr 2021 13:57:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11577 Wed 07 Apr 2021 13:56:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11295 Wed 07 Apr 2021 13:56:38 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11020 Wed 07 Apr 2021 13:56:20 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10768 Wed 07 Apr 2021 13:56:07 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10751 Wed 07 Apr 2021 13:56:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10587 Wed 07 Apr 2021 13:55:55 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10534 Wed 07 Apr 2021 13:55:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10521 0.05. No response to load was seen in either T1DM or control group over the 4-days post exercise. Conclusion: Active individuals with T1DM do not have a heightened Achilles tendon response to load, which suggests no increased risk of tendon injury. We cannot extrapolate these findings to sedentary individuals with T1DM.]]> Wed 07 Apr 2021 13:55:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10524 Wed 07 Apr 2021 13:55:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10354 Wed 07 Apr 2021 13:55:42 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10295 Wed 07 Apr 2021 13:55:38 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10240 Wed 07 Apr 2021 13:55:34 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:9285 Wed 07 Apr 2021 13:54:29 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:8268 Wed 07 Apr 2021 13:53:20 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7635 Wed 07 Apr 2021 13:47:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7610 Wed 07 Apr 2021 13:47:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7615 Wed 07 Apr 2021 13:47:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14555 Tue 30 Nov 2021 09:34:36 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14733 Tue 28 Jun 2022 11:00:01 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15727 Tue 22 Feb 2022 14:46:04 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14703 = 18 years) recruited through opportunistic sampling were screened at sites in 92 countries during May 2019. Ideally, 3 blood pressure readings were measured for each participant, and data on lifestyle factors and comorbidities were collected. Hypertension was defined as a systolic blood pressure >= 140 mm Hg, or a diastolic blood pressure >= 90 mm Hg (mean of the second and third readings) or taking antihypertensive medication. When necessary, multiple imputation was used to estimate participants' mean blood pressure. Mixed-effects models were used to evaluate associations between blood pressure and participant characteristics. Of 1 508 130 screenees 482 273 (32.0%) had never had a blood pressure measurement before and 513 337 (34.0%) had hypertension, of whom 58.7% were aware, and 54.7% were on antihypertensive medication. Of those on medication, 57.8% were controlled to <140/90 mm Hg, and 28.9% to <130/80 mm Hg. Of all those with hypertension, 31.7% were controlled to <140/90 mm Hg, and 350 825 (23.3%) participants had untreated or inadequately treated hypertension. Of those taking antihypertensive medication, half were taking only a single drug, and 25% reported using aspirin inappropriately. This survey is the largest ever synchronized and standardized contemporary compilation of global blood pressure data. This campaign is needed as a temporary substitute for systematic blood pressure screening in many countries worldwide.]]> Tue 19 Oct 2021 16:23:32 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12377 Tue 19 Mar 2024 11:38:09 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12576 Tue 15 Feb 2022 15:56:04 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15527 Tue 14 Dec 2021 10:19:11 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15704 Tue 08 Mar 2022 12:09:41 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14869 Tue 07 Dec 2021 16:02:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15635 Tue 04 Jan 2022 13:35:13 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15609 Tue 04 Jan 2022 11:22:14 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15163 Tue 03 Aug 2021 14:49:31 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13266 Tue 01 Feb 2022 14:27:21 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15182 Thu 26 Aug 2021 12:33:37 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14446 65 years, implemented one or more exercise therapy arms using aerobic, resistance, or mind-body exercise, and assessed depressive symptoms at baseline and follow-up using a validated clinical questionnaire. Results: A network meta-analysis was performed on 15 eligible RCTs comprising 596 participants (321 treatment and 275 controls), including aerobic (n = 6), resistance (n = 5), and mind-body (n = 4) exercise trials. Compared with controls, mind-body exercise showed the largest improvement on depressive symptoms (g = −0.87 to −1.38), followed by aerobic exercise (g = −0.51 to −1.02), and resistance exercise (g = −0.41 to −0.92). Notably, there were no statistically significant differences between exercise types: aerobic versus resistance (g = −0.10, PrI = −2.23, 2.03), mind-body versus aerobic (g = −0.36, PrI = −2.69, 1.97), or mind-body versus resistance (g = −0.46, PrI = −2.75, 1.83). Conclusions: These findings should guide optimal exercise prescription for allied health professionals and stakeholders in clinical geriatrics. Notably, clinically depressed older adults may be encouraged to self-select their preferred exercise type in order to achieve therapeutic benefit on symptoms of depression. In coalition with high levels of compliance, these data provide encouraging evidence for the antidepressant effect of either aerobic, resistance, or mind-body exercise as effective treatment adjucts for older adults presenting with clinical depression. © 2019]]> Thu 25 Nov 2021 11:34:55 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15735 Thu 24 Feb 2022 09:28:04 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14921 Thu 21 Oct 2021 16:29:07 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14485 Thu 21 Oct 2021 16:24:31 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13852 0.05, in both groups). CRP increased in the South Asian group ( p<0.05) but not the Caucasian group ( p>0.05) Conclusions: The cardio-metabolic response to resistance exercise is comparable in young Caucasian and South Asian males though inflammatory response to exercise may be prolonged in South Asians.]]> Thu 19 May 2022 12:43:22 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14223 Thu 19 May 2022 12:38:25 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14617 Thu 19 May 2022 12:02:09 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15266 Thu 19 May 2022 11:39:59 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15458 Thu 18 Nov 2021 16:20:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15417 Thu 18 Nov 2021 10:02:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15799 Thu 17 Mar 2022 12:51:05 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15231 Thu 17 Feb 2022 10:39:03 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15206 Thu 16 Sep 2021 12:10:27 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13324 Thu 16 Dec 2021 12:03:57 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14309 C and angiotensin-converting enzyme insertion and deletion polymorphisms) and biomarkers such as interleukin-6, tumor necrosis factor, soluble (s) E-selectin, homocysteine, and highly sensitive C-reactive protein have been shown to affect blood pressure variability among non-African CKD, hypertensive. and nonhypertensive CKD population. However, the contributions of the pattern, genetic, and environmental determinants of ambulatory blood pressure in African CKD have not been characterized. Understanding these interactions may help to develop interventions to prevent major cardiovascular events among people with CKD. Objective: The overarching objective of this study is to identify, document, and develop approaches to address related phenomic, genetic, and environmental determinants of ambulatory blood pressure patterns in African CKD and non-CKD hypertensive patients compared with normotensive controls. Methods: This is a longitudinal short-term follow-up study of 200 adult subjects with CKD and 200 each of age-matched hypertensives without CKD and apparently healthy controls. Demographic information, detailed clinical profile, electrocardiography, echocardiography, and 24-hr ambulatory blood pressure measurements will be obtained. Blood samples will be collected to determine albumin-creatinine ratio, fasting plasma glucose, lipid profile, electrolytes, urea and creatinine, C-reactive protein, serum homocysteine, fibroblast growth factor-23, and complete blood count, while 2 mL blood aliquot will be collected in EDTA (ethylenediaminetetraacetic acid) tubes and mixed using an electronic rolling system to prevent blood clots and subsequently used for DNA extraction and genetic analysis. Results: A total of 239 participants have been recruited so far, and it is expected that the recruitment phase will be complete in June 2020. The follow-up phase will continue with data analysis and publications of results. Conclusions: This study will help stratify Nigerian CKD patients phenotypically and genotypically in terms of their blood pressure variations with implications for targeted interventions and timing of medications to improve prognosis.]]> Thu 09 Dec 2021 15:35:02 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13014 Thu 07 Oct 2021 16:13:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15395 Thu 04 Nov 2021 10:02:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14477 Thu 03 Feb 2022 12:44:00 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14669 Thu 02 Dec 2021 16:14:24 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15797 Mon 28 Mar 2022 11:43:12 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15365 Mon 25 Oct 2021 14:15:11 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15350 Mon 25 Oct 2021 12:30:11 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15343 Mon 25 Oct 2021 12:16:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15668 Mon 24 Jan 2022 15:39:13 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15660 Mon 24 Jan 2022 14:30:29 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15693 Mon 15 Aug 2022 10:22:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15013 Mon 12 Apr 2021 15:22:09 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11289 Mon 10 May 2021 10:11:54 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15053 Mon 10 Jan 2022 15:12:41 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15105 Mon 09 May 2022 16:48:59 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15710 Mon 07 Feb 2022 16:24:21 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15692 80% had dyslipidaemia and <15% of patients were diagnosed with diabetes (DM). All of these disorders were more frequent in men. In 80% of patients the waist circumference exceed norm for the European population. Less than half of the patients were current smokers or had smoked in the past. Patients with CVD had significantly higher blood pressure and glucose levels but lower low density lipoprotein-cholesterol level. Conclusions: The prevalence of CVD and CV risk factors among patients in Poland is high. CVD is more common in men than in women. The most common CV risk factors are excess waist circumference, dyslipidaemia and HTN. Family physicians should conduct activities to prevent, diagnose early and treat CVD in the primary health care population. © 2021 Elsevier B.V. **Please note that there are multiple authors for this article therefore only the name of the first 5 including Federation University Australia affiliate “Fadi Charchar” is provided in this record**]]> Mon 07 Feb 2022 15:14:21 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15685 Mon 07 Feb 2022 14:39:21 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15390 Mon 01 Nov 2021 14:08:11 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15375 Mon 01 Nov 2021 10:31:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15713 Fri 03 Nov 2023 12:12:48 AEDT ]]>