http://researchonline.federation.edu.au/vital/access/manager/Index ${session.getAttribute("locale")} 5 http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14820 Wed 25 Nov 2020 14:15:41 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14469 Wed 07 Apr 2021 14:02:33 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14277 Wed 07 Apr 2021 14:02:22 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14221 Wed 07 Apr 2021 14:02:18 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14149 Wed 07 Apr 2021 14:02:14 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14048 Wed 07 Apr 2021 14:02:08 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13937 Wed 07 Apr 2021 14:02:02 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13672 Wed 07 Apr 2021 14:01:47 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13501 Wed 07 Apr 2021 14:01:38 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13349 Wed 07 Apr 2021 14:01:30 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13085 Wed 07 Apr 2021 14:01:16 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12914 7 mg/dL in men and >6 mg/dL in women. The association between sUA concentration and SBP and DBP was examined using Pearson's correlation test, multivariate linear regression, and logistic regression analysis. The prevalence of hypertension and HUA increased with age (P < .001). Hypertension was more common in participants that had HUA than in those that did not (38.95% vs 30.16%, P = .02). Higher sUA was significantly associated with higher SBP and DBP in the 41- to 50-year-old participants (SBP, β = 0.35, P < .001; DBP, β = .29, P < .001; after adjustment for age, sex, total cholesterol, estimated glomerular filtration rate, and fasting plasma glucose). HUA was also a risk factor for hypertension in this age group (odds ratio 1.425, 95% confidence interval, 1.217-1.668, P < .001). There was no association between sUA concentration and SBP and DBP in the other age groups. In this population of healthy Chinese participants, sUA concentration was positively associated with hypertension only in the 41- to 50-year-old group. Lowering uric acid in this age group may help to reduce the incidence of hypertension.]]> Wed 07 Apr 2021 14:01:07 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:12915 Wed 07 Apr 2021 14:01:07 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11795 0.04 ng/mL), after performing high-intensity cycle ergometer sprints. As creatine kinase (CK) is expressed by multiple organs (e.g., skeletal muscle, brain, and myocardium), cTnI assays were performed to determine any changes in total serum CK levels not originating from skeletal muscle damage. Diagnosis: A posteriori the individual's daily energy expenditure indicated chronically low-energy availability. Psychometric testing suggested that the individual scored positive for disordered eating, highly for fatigue levels, and low in mental health components. Outcomes: The current case report provides novel evidence of elevated cTnI occurring as a result of performing short duration, high intensity, cycle ergometer exercise in an individual with self-reported chronically depleted energy balance. A schematic to identify potentially “at risk” individuals is presented. Lessons: Considering this as a case report, results cannot be generalized; however, the main findings suggest that individuals who habitually restrict their calorie intake below their bodies’ daily energy requirements, may have elevated biomarkers of exercise induced myocardial stress from performing high-intensity exercise.]]> Wed 07 Apr 2021 13:57:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11671 Wed 07 Apr 2021 13:56:58 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11678 Wed 07 Apr 2021 13:56:58 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11594 Wed 07 Apr 2021 13:56:53 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11574 Wed 07 Apr 2021 13:56:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11573 Wed 07 Apr 2021 13:56:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11481 Wed 07 Apr 2021 13:56:47 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11241 Wed 07 Apr 2021 13:56:35 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11247 Wed 07 Apr 2021 13:56:35 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11246 Wed 07 Apr 2021 13:56:35 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11225 Wed 07 Apr 2021 13:56:33 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11019 Wed 07 Apr 2021 13:56:20 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11020 Wed 07 Apr 2021 13:56:20 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10895 Wed 07 Apr 2021 13:56:16 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10899 Wed 07 Apr 2021 13:56:16 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10852 Wed 07 Apr 2021 13:56:13 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10703 Wed 07 Apr 2021 13:56:03 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10537 Wed 07 Apr 2021 13:55:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10341 Wed 07 Apr 2021 13:55:41 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10240 Wed 07 Apr 2021 13:55:34 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10214 Wed 07 Apr 2021 13:55:33 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10200 Wed 07 Apr 2021 13:55:32 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10213 Wed 07 Apr 2021 13:55:32 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:9603 1000 young apparently healthy white men from the general population. Each Y chromosome was classified into one of 13 most common European lineages. Androgens (DHEA-S, androstenedione, total testosterone) and their metabolites (total estradiol, estrone) were measured by radioimmunoassays. Information on five dimensions of aggression (total, physical, verbal, anger and hostility) was collected using Buss and Perry questionnaire. Results: Approximately 17% men inherited haplogroup I from their fathers. Carriers of haplogroup I showed lower scores of verbal aggression than men with other haplogroups (beta = -0.72, SE = 0.29, P = 0.012) and when further compared to carriers of most common R1a lineage and other haplogroups (beta = -1.03, SE = 0.34, P = 0.003). However, these associations did not survive a correction for multiple testing. Sex steroids did not show even nominal level of association with haplogroup I. Conclusion: Our data show no overall association between haplogroup I and sex-related phenotypes in young white men. These results also suggest that the previously identified association between haplogroup I and coronary artery disease is not likely mediated by unfavourable profile of sex steroids or heightened aggression early in life. (C) 2014 Elsevier Ireland Ltd. All rights reserved.]]> Wed 07 Apr 2021 13:54:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:9284 Wed 07 Apr 2021 13:54:29 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:9285 Wed 07 Apr 2021 13:54:29 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7659 Wed 07 Apr 2021 13:47:08 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7615 Wed 07 Apr 2021 13:47:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7520 Wed 07 Apr 2021 13:46:58 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7521 Wed 07 Apr 2021 13:46:58 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7274 Wed 07 Apr 2021 13:46:45 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7273 Wed 07 Apr 2021 13:46:44 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7270 Wed 07 Apr 2021 13:46:44 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7167 Wed 07 Apr 2021 13:46:40 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7171 Wed 07 Apr 2021 13:46:40 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7179 Wed 07 Apr 2021 13:46:40 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7178 Wed 07 Apr 2021 13:46:40 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7153 Wed 07 Apr 2021 13:46:39 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7161 Wed 07 Apr 2021 13:46:39 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7083 Wed 07 Apr 2021 13:46:36 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6744 Wed 07 Apr 2021 13:46:18 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6623 Wed 07 Apr 2021 13:46:12 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6601 Wed 07 Apr 2021 13:46:10 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6317 Wed 07 Apr 2021 13:45:54 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6316 Wed 07 Apr 2021 13:45:54 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6315 Wed 07 Apr 2021 13:45:54 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:6300 Wed 07 Apr 2021 13:45:53 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:5339 84%). A sample of 270 patients formed the sample for the study of risk assessment scoring accuracy and use of prevention strategies. It was found usual-care nurses under-estimated patients' risk of pressure ulcer development and under-utilised prevention strategies compared with experienced injury prevention nurses. Despite a significant reduction in prevalence of hospital-acquired pressure ulcers and high documentation compliance, use of prevention strategies could further be improved to achieve better patient outcomes. Barriers to the use of prevention strategies by nurses in the acute hospital setting require further examination. This study provides important insights into the knowledge translation of pressure ulcer prevention best practice guideline recommendations at The Northern Hospital.]]> Wed 07 Apr 2021 13:44:58 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:5266 Wed 07 Apr 2021 13:44:54 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:5099 Wed 07 Apr 2021 13:44:43 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:4959 Wed 07 Apr 2021 13:44:33 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:4890 Wed 07 Apr 2021 13:44:27 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:4764 Wed 07 Apr 2021 13:44:18 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:3147 Wed 07 Apr 2021 13:34:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:3145 Wed 07 Apr 2021 13:34:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:3148 Wed 07 Apr 2021 13:34:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:3151 Wed 07 Apr 2021 13:34:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:2909 Wed 07 Apr 2021 13:33:56 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:2507 Wed 07 Apr 2021 13:33:34 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:2460 Wed 07 Apr 2021 13:33:32 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:2418 Wed 07 Apr 2021 13:33:29 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:2407 Wed 07 Apr 2021 13:33:29 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:235 Wed 07 Apr 2021 13:31:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:221 Wed 07 Apr 2021 13:31:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:226 Wed 07 Apr 2021 13:31:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:184 Wed 07 Apr 2021 13:31:02 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:122 Wed 07 Apr 2021 13:30:57 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:127 Wed 07 Apr 2021 13:30:57 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15727 Tue 22 Feb 2022 14:46:04 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14703 = 18 years) recruited through opportunistic sampling were screened at sites in 92 countries during May 2019. Ideally, 3 blood pressure readings were measured for each participant, and data on lifestyle factors and comorbidities were collected. Hypertension was defined as a systolic blood pressure >= 140 mm Hg, or a diastolic blood pressure >= 90 mm Hg (mean of the second and third readings) or taking antihypertensive medication. When necessary, multiple imputation was used to estimate participants' mean blood pressure. Mixed-effects models were used to evaluate associations between blood pressure and participant characteristics. Of 1 508 130 screenees 482 273 (32.0%) had never had a blood pressure measurement before and 513 337 (34.0%) had hypertension, of whom 58.7% were aware, and 54.7% were on antihypertensive medication. Of those on medication, 57.8% were controlled to <140/90 mm Hg, and 28.9% to <130/80 mm Hg. Of all those with hypertension, 31.7% were controlled to <140/90 mm Hg, and 350 825 (23.3%) participants had untreated or inadequately treated hypertension. Of those taking antihypertensive medication, half were taking only a single drug, and 25% reported using aspirin inappropriately. This survey is the largest ever synchronized and standardized contemporary compilation of global blood pressure data. This campaign is needed as a temporary substitute for systematic blood pressure screening in many countries worldwide.]]> Tue 19 Oct 2021 16:23:32 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15609 Tue 04 Jan 2022 11:22:14 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14921 Thu 21 Oct 2021 16:29:07 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14223 Thu 19 May 2022 12:38:25 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15417 Thu 18 Nov 2021 10:02:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15725 Thu 17 Nov 2022 16:45:30 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14309 C and angiotensin-converting enzyme insertion and deletion polymorphisms) and biomarkers such as interleukin-6, tumor necrosis factor, soluble (s) E-selectin, homocysteine, and highly sensitive C-reactive protein have been shown to affect blood pressure variability among non-African CKD, hypertensive. and nonhypertensive CKD population. However, the contributions of the pattern, genetic, and environmental determinants of ambulatory blood pressure in African CKD have not been characterized. Understanding these interactions may help to develop interventions to prevent major cardiovascular events among people with CKD. Objective: The overarching objective of this study is to identify, document, and develop approaches to address related phenomic, genetic, and environmental determinants of ambulatory blood pressure patterns in African CKD and non-CKD hypertensive patients compared with normotensive controls. Methods: This is a longitudinal short-term follow-up study of 200 adult subjects with CKD and 200 each of age-matched hypertensives without CKD and apparently healthy controls. Demographic information, detailed clinical profile, electrocardiography, echocardiography, and 24-hr ambulatory blood pressure measurements will be obtained. Blood samples will be collected to determine albumin-creatinine ratio, fasting plasma glucose, lipid profile, electrolytes, urea and creatinine, C-reactive protein, serum homocysteine, fibroblast growth factor-23, and complete blood count, while 2 mL blood aliquot will be collected in EDTA (ethylenediaminetetraacetic acid) tubes and mixed using an electronic rolling system to prevent blood clots and subsequently used for DNA extraction and genetic analysis. Results: A total of 239 participants have been recruited so far, and it is expected that the recruitment phase will be complete in June 2020. The follow-up phase will continue with data analysis and publications of results. Conclusions: This study will help stratify Nigerian CKD patients phenotypically and genotypically in terms of their blood pressure variations with implications for targeted interventions and timing of medications to improve prognosis.]]> Thu 09 Dec 2021 15:35:02 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15395 Thu 04 Nov 2021 10:02:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15680 Thu 03 Feb 2022 17:26:38 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14477 Thu 03 Feb 2022 12:44:00 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15343 Mon 25 Oct 2021 12:16:10 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13726 0.63 (p=<0.001), respectively, and a single ‘teamwork’ construct was identified. Non-technical skills overall were good but leadership was rated notably lower than task and teamwork performance indicating a need for leadership training. ConclusionThe TEAM is a feasible, valid and reliable non-technical assessment measure in simulated and real clinical settings. Emergency teams need to develop leadership skills through training and reflective debriefing.]]> Mon 15 Aug 2022 10:30:36 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14125 Mon 15 Aug 2022 10:28:27 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:14694 Mon 15 Aug 2022 10:26:03 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15053 Mon 10 Jan 2022 15:12:41 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15105 Mon 09 May 2022 16:48:59 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15685 Mon 07 Feb 2022 14:39:21 AEDT ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:15375 Mon 01 Nov 2021 10:31:10 AEDT ]]>