http://researchonline.federation.edu.au/vital/access/manager/Index ${session.getAttribute("locale")} 5 http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:13241 Wed 07 Apr 2021 14:01:24 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11679 Wed 07 Apr 2021 13:56:58 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:11116 Wed 07 Apr 2021 13:56:26 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10766 Wed 07 Apr 2021 13:56:07 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10738 Wed 07 Apr 2021 13:56:05 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10609 Wed 07 Apr 2021 13:55:57 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10534 Wed 07 Apr 2021 13:55:52 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10529 0.05). Resting heart rate emerged as an independent predictor of leukocyte telomere length and TERT and TPP1 mRNA expression in stepwise regression models. To gauge whether volume of exercise was associated with leukocyte telomere length, we divided subjects into running and cycling tertiles (distance covered per week) and found individuals in the middle and highest tertiles had longer telomeres than individuals in the lowest tertile. These data emphasize the importance of cardiorespiratory fitness and exercise training in the prevention of biological aging. They also support the concept that moderate amounts of exercise training protects against biological aging, while higher amounts may not elicit additional benefits.]]> Wed 07 Apr 2021 13:55:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10465 Wed 07 Apr 2021 13:55:50 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10351 Wed 07 Apr 2021 13:55:42 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:10127 Wed 07 Apr 2021 13:55:26 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:9603 1000 young apparently healthy white men from the general population. Each Y chromosome was classified into one of 13 most common European lineages. Androgens (DHEA-S, androstenedione, total testosterone) and their metabolites (total estradiol, estrone) were measured by radioimmunoassays. Information on five dimensions of aggression (total, physical, verbal, anger and hostility) was collected using Buss and Perry questionnaire. Results: Approximately 17% men inherited haplogroup I from their fathers. Carriers of haplogroup I showed lower scores of verbal aggression than men with other haplogroups (beta = -0.72, SE = 0.29, P = 0.012) and when further compared to carriers of most common R1a lineage and other haplogroups (beta = -1.03, SE = 0.34, P = 0.003). However, these associations did not survive a correction for multiple testing. Sex steroids did not show even nominal level of association with haplogroup I. Conclusion: Our data show no overall association between haplogroup I and sex-related phenotypes in young white men. These results also suggest that the previously identified association between haplogroup I and coronary artery disease is not likely mediated by unfavourable profile of sex steroids or heightened aggression early in life. (C) 2014 Elsevier Ireland Ltd. All rights reserved.]]> Wed 07 Apr 2021 13:54:51 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:7624 Wed 07 Apr 2021 13:47:06 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:5741 Wed 07 Apr 2021 13:45:18 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:5099 Wed 07 Apr 2021 13:44:43 AEST ]]> http://researchonline.federation.edu.au/vital/access/manager/Repository/vital:4526 Wed 07 Apr 2021 13:43:59 AEST ]]>